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Brief Reports
Citations 31-40 of 1234 total displayed.
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Past content
(since Jul 1999):
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Philadelphia-positive patients who already harbor imatinib-resistant Bcr-Abl kinase domain mutations have a higher likelihood of developing additional mutations associated with resistance to second- or third-line tyrosine kinase inhibitors
- Simona Soverini, Alessandra Gnani, Sabrina Colarossi, Fausto Castagnetti, Elisabetta Abruzzese, Stefania Paolini, Serena Merante, Ester Orlandi, Silvia de Matteis, Antonella Gozzini, Ilaria Iacobucci, Francesca Palandri, Gabriele Gugliotta, Cristina Papayannidis, Angela Poerio, Marilina Amabile, Daniela Cilloni, Gianantonio Rosti, Michele Baccarani, and Giovanni Martinelli
Blood 2009; 114: 2168-2171.
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Genetic evidence for a predominant role of PI3Kβ catalytic activity in ITAM- and integrin-mediated signaling in platelets
- Ilaria Canobbio, Lucia Stefanini, Lina Cipolla, Elisa Ciraolo, Cristian Gruppi, Cesare Balduini, Emilio Hirsch, and Mauro Torti
Blood 2009; 114: 2193-2196.
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Definitive proof for direct reprogramming of hematopoietic cells to pluripotency
- Motohito Okabe, Makoto Otsu, Dong Hyuck Ahn, Toshihiro Kobayashi, Yohei Morita, Yukiko Wakiyama, Masafumi Onodera, Koji Eto, Hideo Ema, and Hiromitsu Nakauchi
Blood 2009; 114: 1764-1767.
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Mutations in CBL occur frequently in juvenile myelomonocytic leukemia
- Mignon L. Loh, Debbie S. Sakai, Christian Flotho, Michelle Kang, Manfred Fliegauf, Sophie Archambeault, Charles G. Mullighan, Leslie Chen, Eva Bergstraesser, Carlos E. Bueso-Ramos, Peter D. Emanuel, Henrik Hasle, Jean-Pierre Issa, Marry M. van den Heuvel-Eibrink, Franco Locatelli, Jan Stary, Monica Trebo, Marcin Wlodarski, Marco Zecca, Kevin M. Shannon, and Charlotte M. Niemeyer
Blood 2009; 114: 1859-1863.
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Absence of collagen-induced platelet activation caused by compound heterozygous GPVI mutations
- Bénédicte Dumont, Dominique Lasne, Chantal Rothschild, Maxime Bouabdelli, Véronique Ollivier, Claire Oudin, Nadine Ajzenberg, Bernard Grandchamp, and Martine Jandrot-Perrus
Blood 2009; 114: 1900-1903.
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Naturally processed peptides spanning the HPA-1a polymorphism are efficiently generated and displayed from platelet glycoprotein by HLA-DRB3*0101–positive antigen-presenting cells
- Gholamreza Anani Sarab, Michael Moss, Robert N. Barker, and Stanislaw J. Urbaniak
Blood 2009; 114: 1954-1957.
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Analysis of the Ten-Eleven Translocation 2 (TET2) gene in familial myeloproliferative neoplasms
- Cécile Saint-Martin, Gwendoline Leroy, François Delhommeau, Gérard Panelatti, Sabrina Dupont, Chloé James, Isabelle Plo, Dominique Bordessoule, Christine Chomienne, André Delannoy, Alain Devidas, Martine Gardembas-Pain, Françoise Isnard, Yves Plumelle, Olivier Bernard, William Vainchenker, Albert Najman, Christine Bellanné-Chantelot, and the French Group of Familial Myeloproliferative Disorders
Blood 2009; 114: 1628-1632.
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Association of hereditary thrombocythemia and distal limb defects with a thrombopoietin gene mutation
- Claudio Graziano, Simona Carone, Emanuele Panza, Flora Marino, Pamela Magini, Giovanni Romeo, Andrea Pession, and Marco Seri
Blood 2009; 114: 1655-1657.
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Thiopurine methyltransferase genetics is not a major risk factor for secondary malignant neoplasms after treatment of childhood acute lymphoblastic leukemia on Berlin-Frankfurt-Münster protocols
- Martin Stanulla, Elke Schaeffeler, Anja Möricke, Sally A. Coulthard, Gunnar Cario, André Schrauder, Peter Kaatsch, Michael Dördelmann, Karl Welte, Martin Zimmermann, Alfred Reiter, Michel Eichelbaum, Hansjörg Riehm, Martin Schrappe, and Matthias Schwab
Blood 2009; 114: 1314-1318.
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BIO5192, a small molecule inhibitor of VLA-4, mobilizes hematopoietic stem and progenitor cells
- Pablo Ramirez, Michael P. Rettig, Geoffrey L. Uy, Elena Deych, Matthew S. Holt, Julie K. Ritchey, and John F. DiPersio
Blood 2009; 114: 1340-1343.
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