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Blood, Vol. 111, Issue 5, 2647-2656, March 1, 2008 This Article Abstract Full Text Services Email this article to a friend Alert me to new issues of the journal Rights and Permissions Citing Articles Citing Articles via CrossRef Defective angiogenesis, endothelial migration, proliferation, and MAPK signaling in Rap1b-deficient mice Blood Chrzanowska-Wodnicka et al. 111: 2647 Supplemental materials for: Chrzanowska-Wodnicka et al Files in this Data Supplement: Document 1. Supplemental materials and methods (PDF, 57.0 KB) Figure 1. Characterization of lung microcircular endothelial cells from rap1b−/− mice (JPG, 85.2 KB) - (A) Surface expression of PECAM (left graph) and Flk-1 (right graph) on wild-type (blue line) and Rap1b−/− (brown line) cells. As a control, cells were stained with isotype-matched unrelated control antibodies (wild-type: red line and Rap1b−/−: green line). (B) VE-cadherin immunofluorescent staining of cell-cell junctions in wild-type and Rap1b−/− cells. Bar: 20 µm. (C) Total Rap1 protein expression in wild-type and Rap1b−/− cells. Rap1b is the predominant isoform of Rap1 in mouse lung endothelial cells. Equal protein loading is evidenced by re-probing the blot with anti-actin antibody. This Article Abstract Full Text Services Email this article to a friend Alert me to new issues of the journal Rights and Permissions Citing Articles Citing Articles via CrossRef

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