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Blood, Vol. 112, Issue 2, 406-414, July 15, 2008 This Article Abstract Full Text Services Email this article to a friend Alert me to new issues of the journal Rights and Permissions Citing Articles Citing Articles via CrossRef H3 K79 dimethylation marks developmental activation of the β-globin gene but is reduced upon LCR-mediated high-level transcription Blood Sawado et al. 112: 406 Supplemental materials for: Sawado et al Files in this Data Supplement: Document 1. Supplemental materials and methods (PDF, 93.7 KB) Table S1. Allele-specific analysis of H3 enrichment in the promoter proximal region and third exon in ΔLCR (HbbD) /WT (HbbS) heterozygous mouse spleen cells (PDF, 11.5 KB) - Enrichments at the WT and LCR adult -globin gene relative to myoD1 in sample materials are standardized against those in IgG-bound materials. Each value represents the average of multiple experiments. SD is shown. Table S2. Percentages of c-kit, CD71, and ter-119 positive cells in each GFLC fraction (PDF, 12 KB) - Data obtained from two independent sets of experiments are shown. In both experiments, cells were prestained with anti-c-kit APC, anti-CD71-FITC, and anti-ter119 PE (BD Pharmingen, lot#21115) antibodies before magnetic bead-binding reactions. Each GFLC fraction was analyzed by flow cytometry to identify the populations present in the sorted fractions. This Article Abstract Full Text Services Email this article to a friend Alert me to new issues of the journal Rights and Permissions Citing Articles Citing Articles via CrossRef

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