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Blood, Vol. 113, Issue 4, 883-886, January 22, 2009 This Article Abstract Full Text Services Email this article to a friend Alert me to new issues of the journal Rights and Permissions Citing Articles Citing Articles via CrossRef RUNX1/AML1 DNA-binding domain and ETO/MTG8 NHR2-dimerization domain are critical to AML1-ETO9a leukemogenesis Blood Yan et al. 113: 883 Supplemental materials for: Yan et al Files in this Data Supplement: Figure S1. Unlike AE9a, AE9a-delNHR1, and AE9a-del(350-428), AE9aŽdelNHR2 does not form homodimers (JPG, 60.4 KB) - MigR1 HA-AE9a/MigR1 FLAG-AE9a, MigR1 HA-AE9a-delNHN1/ MigR1 FLAG-AE9a-delNHN1, MigR1 HA-AE9aŽdelNHN2/ MigR1 FLAG-AE9a-delNHN2 or MigR1 HA-AE9a-del(350-428)/ MigR1 FLAG-AE9a-del(350-428) were co-expressed in 293T cells. left panel shows whole cell lysate (WCL) western using HA and FLAG antibody, the right upper panel shows Immunopreciptation (IP) with FLAG and western with HA and the lower right panel shows IP with HA and western with FLAG antibody. Figure S2. The morphology of bone marrow (BM) and spleen (SP) cells from A/E9a del-NHR1 and A/E9a del-350-428 leukemic mice (JPG, 86.1 KB) - The BM and SP cell cytospin slides were stained with Wright-Giemsa solution. Accumulated blast cells were observed in all of the blood tissues from both A/E9a del NHR1 and A/E9a del 350-428 mice. This Article Abstract Full Text Services Email this article to a friend Alert me to new issues of the journal Rights and Permissions Citing Articles Citing Articles via CrossRef

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